Promoting Progressive Healing
Hypochlorous Acid is a Very Potent Broad-Spectrum Antimicrobial Agent
By Randell Varilla, MSN, RN
Advance for Nurses FEATURED article
Posted on: May 23, 2012
Chronic non-healing wounds, such as venous stasis ulcers, diabetic ulcers and pressure ulcers are serious unmet medical needs that effect a patient’s morbidity and mortality.
Factors that inhibit wound closure and healing are bacterial load, biofilm, presence of dead tissue or foreign bodies, deficiency of growth factors, tissue maceration, venous stasis, diabetes, malnutrition, renal disease, and advanced age.
Losing Control
Controlling the bacterial bioburden in wounds has been very difficult.
If the physician cannot control the infection in these chronic wounds, the patient may become further compromised by additional tissue damage, bacteremia, sepsis or deeper wound infections.
Pure Hypochlorous Acid or Electrolyzed Oxidized Water
Hypochlorous acid (HOCl) is a very potent broad-spectrum antimicrobial agent. It is produced by the body’s white blood cells as part of the innate defense against microbial pathogens through phagocytosis and oxidative burst with no-to-low potential for resistance.
In the past, companies and compounding pharmacies have attempted to prepare various forms of pure HOCl unsuccessfully and have unfortunately had to settle with mixtures such as Dakin’s solution, which is a mixture of sodium hypochlorite (bleach) and HOCl (innate immune molecule).
Pure HOCl in solution has been described as being 80-100 times more potent as a germicide than the hypochlorite anion (high pH).
This is because pure HOCl in solution as a neutral/uncharged species can penetrate microbial cells and spore walls while the charged hypochlorite anion cannot penetrate cell walls. Pure HOCl is protonated and neutral in solution at acidic pH’s.
Recent in vitro research studies showed hypochlorous acid was effective against both planktonic and biofilm forms of S. aureus. The researchers concluded the compound was equally effective in vitro against various antibiotic-sensitive and resistant S. aureus isolates when tested in minimum bactericidal concentration and time-kill assays.
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